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Investigation on Sulphide inhibition of anaerobic digestion for Sulphate rich wastewater

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dc.contributor.author Samarathunga, IR
dc.contributor.author Rathnasiri, PG
dc.date.accessioned 2018-12-12T21:30:31Z
dc.date.available 2018-12-12T21:30:31Z
dc.identifier.uri http://dl.lib.mrt.ac.lk/handle/123/13728
dc.description.abstract Skim latex processing wastew iter is enriched with both organic materials and sulfates the environment as it is a nontoxic, non-volatile -Abstract: (SCV2). Sulfate does not cause any direct impact on and chemically inert compound. But biologinil transformation of sulfate under anaerobic conditions produces toxic sulfide, which inhibitory to ethanogenic microorganisms. Toxic, odorous and corrosive free hydrogen sulfide causes reduction in methane yield, significant reduction in overall treatment efficiency or complete failures in anaerobic process. According to the case study conducted, anaerobic reactor of skim latex processing wastewater treatment facility is evaluated.Its daily - wastewater generation is 100m3/day and the average COD and the BOD levels are 5,000 mg/1 and •1000 mg/1 respectively. The COD/SOp2 of the feedstock is 6.25. Although the expected efficiency of the reactor is 50%-80%, the actual COD removal efficiency is only 42% due to sulphide inhibition. Therefore, conventional anaerobic treatment cannot be applicable to skim latex wastewater without taking sufficient precautions to minimize sulfiti 2 inhibition and hydrogen sulfide emission. Microaeration is proposed as an option for treatment c: sulphate rich wastewater. en_US
dc.language.iso en en_US
dc.subject Digestion Methanogenic bacteria en_US
dc.subject Hydrogen sulfide
dc.subject Inhibition
dc.title Investigation on Sulphide inhibition of anaerobic digestion for Sulphate rich wastewater en_US
dc.type Article-Abstract en_US
dc.identifier.year 2015 en_US
dc.identifier.journal Annual Sessions of IESL, The Institution of Engineers, Sri Lanka - 2015 en_US
dc.identifier.issue pt. B en_US
dc.identifier.volume vol. 1 en_US
dc.identifier.pgnos pp. 247 - 255 en_US


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